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I 2 = 45.3%), such as change in diastolic blood pressure (MD: −1.45 mmHg
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105–110 A possible explanation is that pro-inflammatory factors in type 2 diabetes compromise endothelial caveolin-1, a major membrane intrinsic protein in the caveolae on the cell surface, leading to vascular dysfunction, affecting neurogenesis, and subsequently leading to AD
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