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Clinical study suggesting that DPP-4 inhibition blocks distal, rather than proximal, sodium reabsorption after prolonged therapy in patients with T2DM, potentially as a result of decreased inactivation of SDF1 α rather than a mechanism involving GLP-1
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perhaps explained by lixisenatide’s short half-life compared with the long-acting GLP-1RAs being tested in the other CVOTs (Table 1), the short follow-up period of 2 years and the highest percentage of participants on statin therapy (Table 2)
